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The 10 week long experiment generated a homogeneous osteomalacia of vertebral bodies without apparent induction of skeletal malformations. Likewise, scale growth continues with regular annuli formation, but new scale matrix remains without minerals. Bone matrix formation was continuous and uninterrupted, albeit without traces of mineralization. Contrary to the X‐ray‐based diagnosis, the histological examination revealed that vertebral bodies had a regular size and regular internal bone structures intervertebral spaces were not enlarged. On radiographs, vertebral bodies appear small, undersized and with enlarged intervertebral spaces. Bone and scale mineral content decreased by c. Animals that received the P‐deficient diet displayed known signs of P deficiency including reduced growth and soft, pliable opercula. salar at the cellular and at the micro‐anatomical level. This is the first account of how a primary P deficiency affects the skeleton in S. The animal's skeleton was subsequently analysed by radiology, histological examination, histochemical detection of minerals in bones and scales and chemical mineral analysis. The dietary P provision was reduced by 50% for a period of 10 weeks under controlled conditions. To understand the effect of low dietary phosphorus (P) intake on the vertebral column of Atlantic salmon Salmo salar, a primary P deficiency was induced in post‐smolts.